Duration : 0:4:6

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For more, see Dr. Mercola’s site.

http://articles.mercola.com/sites/articles/archive/2008/08/05/fungus-causing-cancer-a-novel-approach-to-the-most-common-form-of-death.aspx

Duration : 0:9:41

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Kris is also the author of two books, “Crazy Sexy Cancer Tips” and “Crazy Sexy Cancer Survivor: More Rebellion and Fire for Your Healing Journey.”

For more info, please stop by
http://www.crazysexycancer.com

Also check out our thriving, groovy online community at http://www.crazysexylife.com

Thanks for watching!

Duration : 0:8:6

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http://norml.org

We also talk about Cannabis in the treatment of ALS (Lou Gehrig’s Disease) and the documentary I’m doing on Cathy Jordan, a medical marijuana patient who has survived ALS for 22 years now!
View “Surviving ALS” in chapter releases:

http://YouTube.com/SurvivingALS

Cannabis Therapeutics Conference hosted by Patients Out of Time:
http://MedicalCannabis.com

Duration : 0:9:30

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http://www.mapinc.org/drugnews/v08/n076/a02.html

On this video filmed in 2006, Dr. Robert Melamede, Professor of Biology at the University of Colorado, explains how the body’s Endo-Cannabinoid system (and plant based Cannabinoids as suppliment) kills cancer cells and inhibits tumor growth.
Refering to 1974 U.S.government-sponsored study when THC was found to slow growth of cancer cells

http://projectcensored.org/publications/2001/22.html

and to recent research in Spain finding that THC stopped brain tumors in rats and protected surrounding nerve tissue, demonstrating neuroprotectant aspect of Cannabinoids.
PDF of Dr. Guzman’s research:

http://americanmarijuana.org/Guzman-Cancer.pdf

Refering to “Homeostatic Regulation” of several body systems by Cannabinoids, Dr. Melamede concludes that Cannabis is indeed a “Miracle Drug”, also recommending hemp oil as excellent source of EFAs the body needs to fight disease.
View Dr. Robert Melamede at 2004 Cannabis Therapeutics Conference:
“Holistic Biochemistry of Cannabinoids”

Dr. Guzman will be at the 2008 Cannabis Therapeutics Conference this April. More info:

http://www.medicalcannabis.com

Stay Tuned!

Duration : 0:7:51

cancer prevention |cancer events|cancer news|cancer

Technorati Tags: alternative, cancer, ganja, leukaemia, lung, lymphoma, marijuana, medical, medicine, prostate, Skin, thyroid, treatment

MEDICAL BIOLOGY: ON TARGETED CANCER THERAPY

1) Oncogene and tumor suppressor gene networks influence the decisions of cancer cells to proliferate or die. These decisions are further influenced by the tumor microenvironment and stress signals, such as DNA damage. Moreover, recent work suggests that a subpopulation of cancer cells with stem-cell-like properties may be critical for triggering tumor development. Together, recent studies have provided a conceptual framework within which practitioners of experimental cancer therapeutics can consider the design of targeted agents.

2) The term “targeted therapy” refers to a new generation of cancer drugs designed to interfere with a specific molecular target (typically a protein) that is believed to have a critical role in tumor growth or progression. The identification of appropriate targets is based on a detailed understanding of the molecular changes underlying cancer. This approach contrasts with the conventional, more empirical approach used to develop cytotoxic chemotherapeutics — the mainstay of cancer drug development in past decades.

3) The clinical success of the small molecule kinase inhibitor imatinib mesylate (Gleevec) in chronic myeloid leukaemia (CML) and gastrointestinal stromal tumors (GIST) has established a paradigm for the treatment of tumors whose growth is acutely dependent on specific kinase targets. CML is driven by the mutant kinase fusion protein Bcr-Abl, which displays constitutive activation of the Abl kinase, whereas GIST is caused by activating point mutations in the c-Kit or platelet derived growth factor receptor (PDGFR)-alpha kinases. Imatinib effectively blocks the activity of all three kinases and produces dramatic clinical responses in all three situations in a manner that correlates precisely with the presence of these mutations in the tumor.

4) In lung cancer, clinical responses to epidermal growth factor receptor (EGFR) inhibitors are associated with point mutations in the EGFR kinase domain (thereby explaining the rather modest 10% response rate in all patients). The clear prediction from this experience is that clinical responses to kinase inhibitors occur in tumors bearing activating mutations that drive tumor progression. Extending this paradigm to larger numbers of cancer patients would require establishing the frequency of kinase mutations in human cancer on a much broader scale — presumably through global gene sequencing efforts analogous to the genome project. Indeed, initial efforts from groups at the Sanger Centre, the Eli Broad Institute, and Johns Hopkins have found previously unsuspected kinase mutations in human tumors.

P53 PROTEIN AS A TARGET IN CANCER THERAPY

1) In 1989, it was discovered that the p53 protein is subtly mutated and thereby inactivated in almost all types of human cancer. These findings raised the possibility that drugs that restore the function of p53 would be broadly applicable cancer treatments. That hope has not yet been realized, but there have been some imaginative approaches to the problem.

2) For 10 years after its discovery in 1979, the p53 gene was thought to be an oncogene. Under normal circumstances, the protein products of oncogenes stimulate appropriate cell division or interfere with cell death. However, certain mutations in these genes result in their products being switched on all the time, or result in gaining a new function. This generally leads to inappropriate stimulation of cell growth, resulting in tumors.

3) Many commonly used drugs inhibit the ability of their target proteins to bind to other molecules or to catalyze reactions, and so mutated oncoproteins are, in theory, perfect drug targets. Theory has been put into practice with the development of STI-571 (Gleevec), a drug that inhibits the aberrantly activated enzyme found in chronic myeloid leukemia cells. But most genetic alterations in common human cancers affect tumor-suppressor genes, rather than oncogenes. The normal function of a tumor suppressor gene is to keep cell numbers down by stopping cells from multiplying or by promoting cell death. When mutated, tumor-suppressor proteins are switched off, and again the result is inappropriate net cell growth.

4) In some cancers, an entire tumor-suppressor gene may be deleted. In general, tumor suppressors are poor targets for conventional drugs, since one cannot inhibit an activity that is not present. Unfortunately, the same work that identified p53 as a potential target for anti-cancer drugs also demonstrated that it behaves genetically like a tumor-suppressor gene and not like an oncogene.

fascinating isn’t it!!

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I could find no evidence of any effective complementary therapies for cervical cancer in PubMed.

A Google search quickly found some suspect looking medical centres, that I would suggest you steer well clear of – this review examines the quality of information on the web for the treatment of cervical cancer: http://www.biomedcentral.com/1472-6874/6/9.

Here are some extracts from a very good review on the topic of nutrition and supplements. The full article is available for free.
“Adequate dietary intake can improve the nutritional status of nearly all cancer survivors…

During active cancer treatment, maintaining caloric balance is the most important nutritional goal. If it is not possible to meet nutritional needs through regular diet alone, nutritious snacks or drinks may be advisable. Commercially prepared liquid nutritional products (e.g., Boost,® Ensure,® Resource,® or NuBasics®) can also be helpful to assist with intake of calories and nutrients. These products are best used as only temporary aids.

Even though cancer treatment can cause fatigue, light, regular physical activity during treatment should be encouraged to improve appetite, stimulate digestion, prevent constipation, maintain energy level and muscle mass, and provide relaxation or stress reduction. Advice from a nutritionally qualified health care provider can be helpful in assessing problems with eating and physical activity and in creating an individualized plan to meet the specific challenges faced during the cancer treatment phase…

It is counterproductive for patients to take vitamin supplements that contain high levels of folic acid, or to eat fortified food products that contain high levels of folic acid when using chemotherapy agents, such as methotrexate, that act by interfering with folic acid metabolism…

Taking antioxidant vitamins during chemotherapy or radiation therapy might … reduce the effectiveness of those therapies, and some experts have therefore recommended against taking antioxidant supplements during such treatment…

Early results from uncontrolled trials suggesting that vitamin C and beta carotene were beneficial for cervical cancer were not supported in a properly controlled trial. And, there is no good evidence that taking supplements of vitamins A, C, or E at doses above the [recommended daily amount] is associated with any clinical benefit”.

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There are no known cures for lung cancer, sorry!

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There are no approved gene therapy treatments for cancer. Research experiments are being done on animals and in vitro (cell cultures) with engineered virus and plasmid vectors to deliver genes to override the oncogenes in cancer cells.

There are problems including how to target the cancer cells, how to prevent an immune response against the virus/plasmid vectors, integration of the new gene into the proper location in the genome and, overcoming a dominant oncogene.

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the Cancer Treatment Centers of America use a holistic approach, mind, body, spirit and incorporate both alternative, nutrition and traditional methods to treat cancer. I do not know if there is one in NY however. Chinese medicine practitioners can probably be found in NY, also just do a web search for natural or holistic health practitioners in your area

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